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"inflammation cannot exist without water"

That's an intriguing statement. But is it true though? "The anti-inflammatory effect of dry fasting is much more powerful than the effect achieved with a water fasting practice. This is because inflammation cannot exist without water. Inflamed areas swell. Micro-organisms, such as microbes and viruses, multiply can only in a sufficiently watery environment. It follows that a water deficit is extremely counterproductive for the inflammatory process. With a DF external water is missing, therefore pathogenic microorganisms immediately find an unfavorable environment. Thanks to the dehydration established by DF, the cells of the body and the pathogenic microorganisms undertake a very strong competition for the appropriation of water. The cells of the body, in their home field, easily manage to seize the water from the microorganisms and not only that. In DF mode the body not only synthesizes endogenous water, but can also acquire it from the air through skin absorption. This is because during a DF the skin stops secreting and shifts to an absorption mode. Healthy and strong cells receive an extra dose of energy and water while the sick ones, viruses and bacteria cannot do it. Microbes, viruses and worms die instantly without water." https://dryfasting.info/what-are-the-advantages-of-dry-fasting-compared-to-water-fasting/

Answer

yeah its true, swelling in my sinuses, inflammatory pain around the spine and nerves (Recurring ischias in particular), swelling in the throat; all of it went away permanently after a few dry fasts that where about 5 days long

It also works on small wounds and acne

Answer

Osmotic stress blocks NF-κB-dependent inflammatory responses by inhibiting ubiquitination of IκB

>The use of hypertonic solutions to reduce acute or chronic inflammation in some pathogenic conditions potentially has benefits, because it could be an inexpensive and safer alternative to anti-inflammatory drugs. Our results demonstrate that osmotic stress can block IL-1-induced activation of NF-κB by inhibiting ubiquitination of IκBα, which inhibits subsequent upregulation of inflammatory cytokines.